Researchers modeled pancreatic cancer development by labeling pancreatic cells with complete mutation (green), incomplete mutation (yellow), or no mutation (red) of the tumor suppressor gene p53, the most commonly mutated gene in cancer. By tracking the green cells that eventually become tumors, they can identify the origins of pancreatic cancer development and the behavior of these cells during tumor formation. Credit: Mandar Deepak Muzumdar
Our annual and topical symposia bring researchers, industry professionals, and clinicians together across disciplines and institutions to explore current trends and knowledge in cancer research. Showcasing the latest discoveries and perspectives through scientific talks, panel discussions, and networking exchanges, these day-long and multi-day events invite discourse and discovery at the leading edge of academic, biopharmaceutical, and clinical practice.